Science moves on the right questions. A gathering of leading aging researchers tried to define those questions from scratch. What emerged says a great deal about where the field stands today.
Some people reach a hundred in reasonable health. Others decline sharply in their seventies. A new review asks why, and the answer is more complicated than most longevity research suggests.
What if aging is not a biological puzzle but a physics problem? A growing group of researchers is applying the laws of physics to the aging process.
Aged skin heals more slowly. That has long been known. But a new experiment shows that removing damaged cells can significantly speed up that process.
Blue zones are held up as proof that certain environments make people live longer. But how solid is the data behind that idea?
Measuring tissue aging usually requires a biopsy or imaging scan. A newly developed probe could make it as simple as collecting urine after a single injection.
Detecting the buildup of aging cells in the body has always required invasive tissue samples. A new study suggests a simple urine test could do the job instead.
Lung disease kills more people than most realize. And aging is the silent driving force behind nearly all of it — even when the immediate cause appears to come from outside.
Astronauts returning from space look as though they have aged by years. That is not coincidence.
A researcher at Rockefeller University has mapped how aging unfolds across nearly seven million cells from 21 mouse tissues at three different ages.
A shark older than five hundred years, swimming through waters it has known since before the Industrial Revolution.
A three-year-old mouse is near the end of its life. A thirty-year-old naked mole-rat looks barely different from how it did at five.
Every cell in your body lives inside a kind of scaffolding — a web of proteins that provides structure, relays signals, and helps determine whether a cell stays healthy or drifts into…
April 2026 was a busy month for researchers treating aging as a biological problem to be solved.
Millions of people are already experimenting with senolytic treatments to slow aging. A new study now shows that one of the most popular combinations causes brain damage strikingly similar to multiple sclerosis…
April 2026 was a busy month for longevity science. New findings, clinical progress and policy shifts — a look at where the field stands. Lifespan.
The field of anti-aging research moves fast. April 2026 brought a wave of new results, company announcements, and scientific insights.
COPD — the lung disease that slowly robs people of their breath — has long been considered irreversible.
Dasatinib and quercetin — the most widely used senolytic combination in longevity circles — causes damage to specific brain regions in mice that closely resembles what is seen in multiple sclerosis.
Conferences are filling up, startups are raising money, and the first human trials of treatments aimed at reversing biological aging are underway.
In a mouse model of spinal disc degeneration, a cheap, decades-old drug combination beat a newer and more expensive competitor.
Not all ant colonies build at the same pace. New experiments show that the age structure of a colony — how many young versus old workers it contains — directly determines how…
Cells that have stopped dividing and are normally considered a hallmark of aging turn out to play a crucial role in wound healing.
A roundup from the Lifespan Research Institute surveys what happened in rejuvenation science over the first months of 2026.
Aging research in Europe is well-funded and growing fast. But scientists in different countries are largely working in isolation, duplicating efforts and missing insights that only emerge from sharing data at scale…
In longevity science, the word ‘breakthrough’ gets used a lot. A recent roundup from Lifespan.io surveys the state of the field.
Two of the most discussed phenomena in aging research — chronic low-grade inflammation and the biological clock of cells — are more tightly linked than previously understood.
It sounds counterintuitive: slightly damaging your cells to make them stronger. Yet that’s precisely the principle behind one of the most intriguing ideas in aging research.
A drug used for decades to treat a rare blood cancer turns out to also clear senescent cells — the so-called zombie cells associated with aging and age-related disease.
Bacterial cells can radically shift their internal molecular composition in response to changing conditions. Yet something remains surprisingly constant.
Pulmonary fibrosis is one of the most devastating age-related diseases — the lungs stiffen progressively, with no effective cure.
Fat tissue ages faster than most other parts of the body. Scientists have now identified the enzyme that keeps that process in check — and what goes wrong when it disappears.
Ageing cells that refuse to die accumulate in our bodies and cause widespread damage. A new drug can now selectively eliminate them in humans — the first data are in.
Fat tissue ages just like the rest of the body. But why does it deteriorate faster in some people?
From a vital aging research database that risks disappearing for lack of funding, to promising animal experiments with plasma exchange and thyroid intervention — March 2026 offered a wide range of developments…
Fat tissue ages. That much has been known for a while. But what drives the aging process inside individual fat cells has remained murky.
A biotech company targeting so-called zombie cells — cells that stop dividing but refuse to die and instead damage surrounding tissue — has shown for the first time that its approach appears…
Animal studies confirm that high doses of nanoplastic particles cause tissue dysfunction.
People are living longer. They are not necessarily living better. The Buck Institute for Research on Aging has announced a new initiative specifically aimed at measuring and extending the years of life…
Senolytics — drugs that selectively eliminate aged, dysfunctional cells — are among the most closely watched interventions in aging research. Until now, the leading candidates have been synthetic compounds.
A biotech company targeting senescent cells has reported positive preliminary results from its first human trial.
Cellular senescence has become one of the most active frontiers in longevity science.
Senolytics — drugs that selectively eliminate aged, dysfunctional cells — are among the most closely watched interventions in aging research. Until now, the leading candidates have been synthetic compounds.
The hunt for safe senolytics — drugs that selectively eliminate aged, dysfunctional cells — has largely focused on repurposed cancer medications.
People are living longer. They are not necessarily living better. The Buck Institute for Research on Aging has announced a new initiative specifically aimed at measuring and extending the years of life…